Β-amyloid(1-42)-induced cholinergic lesions in rat nucleus basalis bidirectionally modulate serotonergic innervation of the basal forebrain and cerebral cortex
Harkany, Tibor O'Mahony, Sheila Keijser, Jan Kelly, John P. Kónya, Csaba Borostyánkői, Zsolt A. Görcs, Tamás J. Zarándi, Márta Penke, Botond Leonard, Brian E.
Harkany, Tibor
O'Mahony, Sheila
Keijser, Jan
Kelly, John P.
Kónya, Csaba
Borostyánkői, Zsolt A.
Görcs, Tamás J.
Zarándi, Márta
Penke, Botond
Leonard, Brian E.
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Publication Date
2001-08-01
Keywords
Externally hosted open access publications with University of Galway authors, anxiety, magnocellular nucleus basalis, serotonin, sprouting, amyloid precursor protein, ca2+ antagonist nimodipine, alzheimers-disease, raphe dorsalis, anxiety disorders, calcium-channels, cortical-neurons, nmda receptor, beta-protein, hippocampus
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journal article
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Harkany, Tibor; O'Mahony, Sheila; Keijser, Jan; Kelly, John P. Kónya, Csaba; Borostyánkői, Zsolt A.; Görcs, Tamás J.; Zarándi, Márta; Penke, Botond; Leonard, Brian E.; Luiten, Paul G.M. (2001). Β-amyloid(1-42)-induced cholinergic lesions in rat nucleus basalis bidirectionally modulate serotonergic innervation of the basal forebrain and cerebral cortex. Neurobiology of Disease 8 (4), 667-678
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Elsevier BV